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A New KIT Mutation (N505I) in Acral Melanoma Confers Constitutive Signaling, Favors Tumorigenic Properties, and Is Sensitive to Imatinib

Maryline Allegra 1 Damien Giacchero 2 Coralie Segalen 1 Nicolas Dumaz 3 Catherine Butori 4 Véronique Hofman 5 Paul Hofman 5 Jean-Philippe Lacour 6 Corine Bertolotto 7 Philippe Bahadoran 7 Robert Ballotti 7 
1 C3M - Centre méditerranéen de médecine moléculaire
2 Medical Oncology Department [Nice]
3 IMRB - Institut Mondor de recherche biomédicale
4 Laboratory of Clinical and Experimental Pathology
5 IRCAN - Institut de Recherche sur le Cancer et le Vieillissement
6 Centre de référence de dermatologie pédiatrique
7 C3M - Centre méditérannéen de médecine moléculaire
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https://www.hal.inserm.fr/inserm-02530592
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Submitted on : Friday, April 3, 2020 - 10:05:54 AM
Last modification on : Saturday, June 25, 2022 - 11:43:39 PM

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INSERM | UPEC-UPEM | CNRS | IMRB | UPEC | UNIV-COTEDAZUR | C3M

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Maryline Allegra, Damien Giacchero, Coralie Segalen, Nicolas Dumaz, Catherine Butori, et al.. A New KIT Mutation (N505I) in Acral Melanoma Confers Constitutive Signaling, Favors Tumorigenic Properties, and Is Sensitive to Imatinib. Journal of Investigative Dermatology, Nature Publishing Group, 2014, 134 (5), pp.1473-1476. ⟨10.1038/jid.2013.525⟩. ⟨inserm-02530592⟩

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