Value of genetic models in understanding the cause and mechanisms of Parkinson's disease, Curr Neurol Neurosci Rep, vol.8, pp.288-296, 2008. ,
Functional models of Parkinson's disease: a valuable tool in the development of novel therapies, Ann Neurol, vol.64, pp.16-29, 2008. ,
Animal models of Parkinson's disease progression, Acta Neuropathol, vol.115, pp.385-398, 2008. ,
New animal models of Parkinson's disease, Mov Disord, vol.26, pp.1198-1205, 2011. ,
Animal models of Parkinson's disease: Limits and relevance to neuroprotection studies, Mov Disord, 2012. ,
The development of small primate models for aging research, ILAR J, vol.52, pp.78-88, 2011. ,
The grey mouse lemur: a non-human primate model for ageing studies, Ageing Res Rev, vol.11, pp.150-162, 2012. ,
URL : https://hal.archives-ouvertes.fr/hal-00650159
Microcebus murinus: a useful primate model for human cerebral aging and Alzheimer's disease?, Genes Brain Behav, vol.5, pp.120-130, 2006. ,
Senile plaques and neurofibrillary changes in the brain of an aged lemurian primate, Microcebus murinus, Neurobiol Aging, vol.13, pp.99-105, 1992. ,
URL : https://hal.archives-ouvertes.fr/hal-01977747
Identification of amyloid beta protein in the brain of the small, short-lived lemurian primate Microcebus murinus, Neurobiol Aging, vol.15, pp.215-220, 1994. ,
URL : https://hal.archives-ouvertes.fr/hal-01977743
Age-associated cerebral atrophy in mouse lemur primates, Neurobiol Aging, vol.32, pp.894-906, 2011. ,
URL : https://hal.archives-ouvertes.fr/mnhn-02292134
Aging affects executive functions and memory in mouse lemur primates, Exp Gerontol, vol.42, pp.223-232, 2007. ,
MRI description of cerebral atrophy in mouse lemur primates, Neurobiol Aging, vol.21, pp.81-88, 2000. ,
Cerebral T2-weighted signal decrease during aging in the mouse lemur primate reflects iron accumulation, Neurobiol Aging, vol.19, pp.65-69, 1998. ,
Role of post-translational modifications in modulating the structure, function and toxicity of alphasynuclein: implications for Parkinson's disease pathogenesis and therapies, Prog Brain Res, vol.183, pp.115-145, 2010. ,
Phosphorylation of Ser-129 is the dominant pathological modification of alpha-synuclein in familial and sporadic Lewy body disease, J Biol Chem, vol.281, pp.29739-29752, 2006. ,