O. Varennes, I. Six, R. Mentaverri, and S. Kamel, Mécanismes physiopathologiques du rétrécissement aortique calcifié. Revue Francophone des, Laboratoires, vol.2017, issue.493, pp.25-32, 2017.

G. W. Eveborn, H. Schirmer, P. Lunde, G. Heggelund, J. B. Hansen et al., Assessment of risk factors for developing incident aortic stenosis: the Tromsø Study, Eur J Epidemiol, vol.29, issue.8, pp.567-75, 2014.

H. Baumgartner, V. Falk, J. J. Bax, D. Bonis, M. Hamm et al., ESC/ EACTS guidelines for the management of valvular heart disease, Rev Esp Cardiol (Engl Ed), vol.71, issue.2, p.110, 2017.

N. M. Rajamannan, F. J. Evans, E. Aikawa, K. J. Grande-allen, L. L. Demer et al., Calcific aortic valve disease: not simply a degenerative process: a review and agenda for research from the National Heart and Lung and Blood Institute Aortic Stenosis Working Group. Executive Summary: calcific aortic valve disease-2011 update, Circulation, vol.124, issue.16, pp.1783-91, 2011.

J. D. Miller, R. M. Weiss, and D. D. Heistad, Calcific aortic valve stenosis: methods, models, and mechanisms, Circ Res, vol.108, issue.11, pp.1392-412, 2011.

K. L. Sider, M. C. Blaser, and C. A. Simmons, Animal models of calcific aortic valve disease, Int J Inflam, p.364310, 2011.

M. Guerraty, M. Iii, and E. R. , Models of aortic valve calcification, J Investig Med, vol.55, issue.6, pp.278-83, 2007.

C. J. Barrick, R. B. Roberts, M. Rojas, N. M. Rajamannan, C. B. Suitt et al., Reduced EGFR causes abnormal valvular differentiation leading to calcific aortic stenosis and left ventricular hypertrophy in C57BL/6J but not 129S1/ SvImJ mice, Am J Physiol Heart Circ Physiol, vol.297, issue.1, pp.65-75, 2009.

G. P. Hajj, Y. Chu, D. D. Lund, J. A. Magida, N. D. Funk et al., Spontaneous aortic regurgitation and valvular cardiomyopathy in mice, Arterioscler Thromb Vasc Biol, vol.35, issue.7, pp.1653-62, 2015.

M. C. Drolet, M. Arsenault, and J. Couet, Experimental aortic valve stenosis in rabbits, J Am Coll Cardiol, vol.41, issue.7, pp.1211-1218, 2003.

M. Scatena, M. F. Jackson, M. Y. Speer, E. M. Leaf, M. C. Wallingford et al., Increased calcific aortic valve disease in response to a diabetogenic, procalcific diet in the LDLr -/-ApoB 100/100 mouse model, Cardiovasc Pathol, vol.34, pp.28-37, 2018.

B. Roosens, S. Droogmans, J. Hostens, J. Somja, E. Delvenne et al., Integrated backscatter for the in vivo quantification of supraphysiological vitamin D 3 -induced cardiovascular calcifications in rats, Cardiovasc Toxicol, vol.11, issue.3, pp.244-52, 2011.

R. F. Chun, I. Hernandez, R. Pereira, L. Swinkles, T. Huijs et al., Differential responses to vitamin D 2 and vitamin D 3 are associated with variations in free 25-hydroxyvitamin D, Endocrinology, vol.157, issue.9, pp.3420-3450, 2016.

J. F. Crocker, S. F. Muhtadie, D. C. Hamilton, and D. E. Cole, The comparative toxicity of vitamin D metabolites in the weanling mouse, Toxicol Appl Pharmacol, vol.80, issue.1, pp.119-145, 1985.

I. N. Sergeev and Q. Song, High vitamin D and calcium intakes reduce diet-induced obesity in mice by increasing adipose tissue apoptosis, Mol Nutr Food Res, vol.58, issue.6, pp.1342-1350, 2014.

B. Roosens, G. Bala, S. Droogmans, G. Van-camp, J. Breyne et al., Animal models of organic heart valve disease, Int J Cardiol, vol.165, issue.3, pp.398-409, 2013.

M. C. Drolet, E. Roussel, Y. Deshaies, J. Couet, and M. Arsenault, A high fat/high carbohydrate diet induces aortic valve disease in C57BL/6J mice, J Am Coll Cardiol, vol.47, issue.4, pp.850-855, 2006.

A. Valcour, C. Zierold, A. L. Podgorski, G. T. Olson, J. V. Wall et al., A novel, fully-automated, chemiluminescent assay for the detection of 1,25-dihydroxyvitamin D in biological samples, J Steroid Biochem Mol Biol, vol.164, pp.120-126, 2016.

O. Henri, C. Pouehe, M. Houssari, L. Galas, L. Nicol et al., Selective stimulation of cardiac lymphangiogenesis reduces myocardial edema and fibrosis leading to improved cardiac function following myocardial infarction, Circulation, vol.133, issue.15, pp.1484-97, 2016.
URL : https://hal.archives-ouvertes.fr/inserm-02296561

K. Tanaka, M. Sata, D. Fukuda, Y. Suematsu, N. Motomura et al., Ageassociated aortic stenosis in apolipoprotein E-deficient mice, J Am Coll Cardiol, vol.46, issue.1, pp.134-175, 2005.

L. Quang, K. Bouchareb, R. Lachance, D. Laplante, M. A. et al., Early development of calcific aortic valve disease and left ventricular hypertrophy in a mouse model of combined dyslipidemia and type 2 diabetes mellitus, Arterioscler Thromb Vasc Biol, vol.34, issue.10, pp.2283-91, 2014.

R. M. Weiss, M. Ohashi, J. D. Miller, S. G. Young, and D. D. Heistad, Calcific aortic valve stenosis in old hypercholesterolemic mice, Circulation, vol.114, pp.2065-2074, 2006.

S. Honda, T. Miyamoto, T. Watanabe, T. Narumi, S. Kadowaki et al., A novel mouse model of aortic valve stenosis induced by direct wire injury, Arterioscler Thromb Vasc Biol, vol.34, issue.2, pp.270-278, 2014.

D. T. Ngo, I. Stafford, D. J. Kelly, A. L. Sverdlov, R. D. Wuttke et al., Vitamin D(2) supplementation induces the development of aortic stenosis in rabbits: interactions with endothelial function and thioredoxin-interacting protein, Eur J Pharmacol, vol.590, issue.1-3, pp.290-296, 2008.

N. Niederhoffer, Y. V. Bobryshev, I. Lartaud-idjouadiene, P. Giummelly, and J. Atkinson, Aortic calcification produced by vitamin D 3 plus nicotine, J Vasc Res, vol.34, issue.5, pp.386-98, 1997.

R. Xu, M. Zhao, Y. Yang, Z. Huang, C. Shi et al., MicroRNA-449c-5p inhibits osteogenic differentiation of human VICs through Smad4-mediated pathway, Sci Rep, vol.7, issue.1, p.8740, 2017.