Delayed graft function in kidney transplantation, Lancet, vol.364, p.1814, 2004. ,
Marked variation in the definition and diagnosis of delayed graft function: a systematic review, Nephrol Dial Transplant, vol.23, p.2995, 2008. ,
Delayed graft function of more than six days strongly decreases long-term survival of transplanted kidneys, Kidney Int, vol.54, p.972, 1998. ,
Delayed graft function and the risk for death with a functioning graft, J Am Soc Nephrol, vol.21, p.153, 2010. ,
Association between delayed graft function and allograft and patient survival: a systematic review and meta-analysis, Nephrol Dial Transplant, vol.24, p.1039, 2009. ,
New cellular and molecular immune pathways in ischemia/reperfusion injury, Am J Transplant, vol.6, p.652, 2006. ,
Delayed graft function in the kidney transplant, Am J Transplant, vol.11, p.2279, 2011. ,
The biology of VEGF and its receptors, Nat Med, vol.9, p.669, 2003. ,
Identification of ligand-induced proteolytic cleavage and ectodomain shedding of VEGFR-1/FLT1 in leukemic cancer cells, Cancer Res, vol.69, p.2607, 2009. ,
Soluble endoglin and other circulating antiangiogenic factors in preeclampsia, N Engl J Med, vol.355, p.992, 2006. ,
Angiogenic factors and preeclampsia, Semin Nephrol, vol.31, p.33, 2011. ,
The soluble VEGF receptor sFlt1 contributes to endothelial dysfunction in CKD, J Am Soc Nephrol, vol.20, p.2235, 2009. ,
Elevated soluble Flt1 inhibits endothelial repair in PR3-ANCA-associated vasculitis, J Am Soc Nephrol, vol.23, p.155, 2012. ,
Complement-mediated inhibition of neovascularization reveals a point of convergence between innate immunity and angiogenesis, Blood, vol.116, p.4395, 2010. ,
Complement activation induces dysregulation of angiogenic factors and causes fetal rejection and growth restriction, J Exp Med, vol.203, p.2165, 2006. ,
C3a and C5a promote renal ischemiareperfusion injury, J Am Soc Nephrol, vol.23, p.1474, 2012. ,
Decreasing plasma soluble IL-1 receptor antagonist and increasing monocyte activation early posttransplant may be involved in pathogenesis of delayed graft function in renal transplant recipients, Clin Transplant, vol.24, p.415, 2010. ,
Intrarenal cells, not bone marrow-derived cells, are the major source for regeneration in postischemic kidney, J Clin Invest, vol.115, p.1756, 2005. ,
Renal ischemic injury results in permanent damage to peritubular capillaries and influences longterm function, Am J Physiol Renal Physiol, vol.281, p.887, 2001. ,
MMP-9 gene deletion mitigates microvascular loss in a model of ischemic acute kidney injury, Am J Physiol Renal Physiol, vol.301, p.101, 2011. ,
Early loss of peritubular capillaries after kidney transplantation, J Am Soc Nephrol, vol.22, p.1024, 2011. ,
Chronic renal hypoxia after acute ischemic injury: effects of L-arginine on hypoxia and secondary damage, Am J Physiol Renal Physiol, vol.284, p.338, 2003. ,
Renal ischemia reperfusion inhibits VEGF expression and induces ADAMTS-1, a novel VEGF inhibitor, Am J Physiol Renal Physiol, vol.294, p.928, 2008. ,
Reduction of circulating soluble Fms-like tyrosine kinase-1 plays a significant role in renal dysfunctionassociated aggravation of atherosclerosis, Circulation, vol.120, p.2470, 2009. ,
Molecular and immunohistochemical characterization of the onset and resolution of human renal allograft ischemia-reperfusion injury, Transplantation, vol.74, p.916, 2002. ,
Differential early posttransplant cytokine responses in living and cadaver donor renal allografts, Transplantation, vol.75, p.1351, 2003. ,
The endothelial cell in ischemic acute kidney injury: implications for acute and chronic function, Kidney Int, vol.72, p.151, 2007. ,
VEGF-121 preserves renal microvessel structure and ameliorates secondary renal disease following acute kidney injury, Am J Physiol Renal Physiol, vol.295, p.1648, 2008. ,
Pilot study of extracorporeal removal of soluble Fms-like tyrosine kinase 1 in preeclampsia, Circulation, vol.124, p.940, 2011. ,
Peritubular capillaries are rarefied in congenital nephrotic syndrome of the Finnish type, Kidney Int, vol.75, p.1099, 2009. ,