BDNF-Dependent Plasticity Induced by Peripheral Inflammation in the Primary Sensory and the Cingulate Cortex Triggers Cold Allodynia and Reveals a Major Role for Endogenous BDNF As a Tuner of the Affective Aspect of Pain

K. Thibault; K. Lin; Armelle Rancillac; M. Fan; T. Snollaerts; V. Sordoillet; M. Hamon; M. Smith; Z. Lenkei; S. Pezet

doi:10.1523/JNEUROSCI.0860-14.2014

BDNF-Dependent Plasticity Induced by Peripheral Inflammation in the Primary Sensory and the Cingulate Cortex Triggers Cold Allodynia and Reveals a Major Role for Endogenous BDNF As a Tuner of the Affective Aspect of Pain

K. Thibault K. Lin Armelle Rancillac ¹ M. Fan T. Snollaerts V. Sordoillet M. Hamon M. Smith Z. Lenkei S. Pezet

1 CIRB - Centre interdisciplinaire de recherche en biologie

Document type :

Journal articles

Domain :

Life Sciences [q-bio]

Life Sciences [q-bio] / Neurons and Cognition [q-bio.NC]

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https://www.hal.inserm.fr/inserm-02119514
Contributor : Armelle Rancillac <>
Submitted on : Friday, May 3, 2019 - 6:17:29 PM
Last modification on : Thursday, May 30, 2019 - 7:18:04 PM

BDNF-Dependent Plasticity Induced by Peripheral Inflammation in the Primary Sensory and the Cingulate Cortex Triggers Cold Allodynia and Reveals a Major Role for Endogenous BDNF As a Tuner of the Affective Aspect of Pain

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BDNF-Dependent Plasticity Induced by Peripheral Inflammation in the Primary Sensory and the Cingulate Cortex Triggers Cold Allodynia and Reveals a Major Role for Endogenous BDNF As a Tuner of the Affective Aspect of Pain

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