T. Colborn, F. Saal, and A. Soto, Developmental effects of endocrine-disrupting chemicals in wildlife and humans, Environmental Health Perspectives, vol.101, issue.5, pp.378-84, 1993.
DOI : 10.1289/ehp.93101378

T. Colborn and C. Clement, Chemically induced alterations in sexual and functional development: the human/wildlife connection, Wingspread consensus statement, pp.1-8, 1992.

J. Toppari, J. Larsen, and P. Christiansen, Male reproductive health and environmental xenoestrogens, Environmental Health Perspectives, vol.104, issue.Suppl 4, pp.741-803, 1996.
DOI : 10.1289/ehp.96104s4741

A. Herbst, Clear cell adenocarcinoma and the current status of DES-exposed females, Cancer, vol.33, issue.S1, pp.484-492, 1997.
DOI : 10.1002/1097-0142(19810715)48:1+<484::AID-CNCR2820481308>3.0.CO;2-X

J. Palmer, L. Wise, and E. Hatch, Prenatal Diethylstilbestrol Exposure and Risk of Breast Cancer, Cancer Epidemiology Biomarkers & Prevention, vol.15, issue.8, pp.1509-1523, 2006.
DOI : 10.1158/1055-9965.EPI-06-0109

D. Barker, The Developmental Origins of Adult Disease, Journal of the American College of Nutrition, vol.20, issue.sup6, pp.733-739, 2003.
DOI : 10.1080/07315724.2004.10719428

E. Dodds and W. Lawson, Synthetic ??strogenic Agents without the Phenanthrene Nucleus, Nature, vol.137, issue.3476, p.996, 1936.
DOI : 10.1038/137996a0

A. Krishnan, P. Stathis, and S. Permuth, Bisphenol A: an estrogenic substance is released from polycarbonate flasks during autoclaving, Endocrinology, vol.132, pp.2279-86, 1993.

A. Calafat, Z. Kuklenyik, and J. Reidy, Urinary Concentrations of Bisphenol A and 4-Nonylphenol in a Human Reference Population, Environmental Health Perspectives, vol.113, issue.4, pp.391-396, 2005.
DOI : 10.1289/ehp.7534

L. Vandenberg, R. Hauser, and M. Marcus, Human exposure to bisphenol A (BPA), Reproductive Toxicology, vol.24, issue.2, pp.139-77, 2007.
DOI : 10.1016/j.reprotox.2007.07.010

V. Saal, F. Hughes, and C. , An Extensive New Literature Concerning Low-Dose Effects of Bisphenol A Shows the Need for a New Risk Assessment, Environmental Health Perspectives, vol.113, issue.8, pp.926-959, 2005.
DOI : 10.1289/ehp.7713

I. Lang, T. Galloway, and A. Scarlett, Association of Urinary Bisphenol A Concentration With Medical Disorders and Laboratory Abnormalities in Adults, JAMA, vol.300, issue.11, pp.1303-1313, 2008.
DOI : 10.1001/jama.300.11.1303

L. Vandenberg, M. Maffini, and C. Sonnenschein, Bisphenol-A and the Great Divide: A Review of Controversies in the Field of Endocrine Disruption, Endocrine Reviews, vol.30, issue.1, pp.75-95, 2009.
DOI : 10.1210/er.2008-0021

B. Rubin, M. Murray, and D. Damassa, Perinatal Exposure to Low Doses of Bisphenol A Affects Body Weight, Patterns of Estrous Cyclicity, and Plasma LH Levels, Environmental Health Perspectives, vol.109, issue.7, pp.675-80, 2001.
DOI : 10.1289/ehp.01109675

E. Somm, V. Schwitzgebel, and A. Toulotte, Perinatal Exposure to Bisphenol A Alters Early Adipogenesis in the Rat, Environmental Health Perspectives, vol.117, issue.10, pp.1549-55, 2009.
DOI : 10.1289/ehp.11342

K. Ryan, A. Haller, and J. Sorrell, Perinatal Exposure to Bisphenol-A and the Development of Metabolic Syndrome in CD-1 Mice, Endocrinology, vol.151, issue.6, pp.2603-2615, 2010.
DOI : 10.1210/en.2009-1218

K. Howdeshell, A. Hotchkiss, and K. Thayer, Exposure to bisphenol A advances puberty, Nature, vol.401, pp.763-767, 1999.

R. Newbold, E. Padilla-banks, and R. Snyder, Perinatal exposure to environmental estrogens and the development of obesity, Molecular Nutrition & Food Research, vol.49, issue.7, pp.912-919, 2007.
DOI : 10.1002/mnfr.200600259

P. Alonso-magdalena, S. Morimoto, and C. Ripoll, The Estrogenic Effect of Bisphenol A Disrupts Pancreatic ??-Cell Function In Vivo and Induces Insulin Resistance, Environmental Health Perspectives, vol.114, issue.1, pp.106-118, 2006.
DOI : 10.1289/ehp.8451

R. Newbold, E. Padilla-banks, and R. Snyder, Developmental exposure to endocrine disruptors and the obesity epidemic, Reproductive Toxicology, vol.23, issue.3, pp.290-296, 2007.
DOI : 10.1016/j.reprotox.2006.12.010

H. Masuno, T. Kidani, and K. Sekiya, Bisphenol A in combination with insulin can accelerate the conversion of 3T3-L1 fibroblasts to adipocytes, J Lipid Res, vol.43, pp.676-84, 2002.

K. Wada, H. Sakamoto, and K. Nishikawa, Life Style-Related Diseases of the Digestive System: Endocrine Disruptors Stimulate Lipid Accumulation in Target Cells Related to Metabolic Syndrome, Journal of Pharmacological Sciences, vol.105, issue.2, pp.133-140, 2007.
DOI : 10.1254/jphs.FM0070034

R. Sargis, D. Johnson, and R. Choudhury, Environmental Endocrine Disruptors Promote Adipogenesis in the 3T3-L1 Cell Line through Glucocorticoid Receptor Activation, Obesity, vol.43, issue.7, pp.1283-1291, 2010.
DOI : 10.1210/jcem-67-6-1122

E. Hugo, T. Brandebourg, and J. Woo, Bisphenol A at Environmentally Relevant Doses Inhibits Adiponectin Release from Human Adipose Tissue Explants and Adipocytes, Environmental Health Perspectives, vol.116, issue.12, pp.1642-1649, 2008.
DOI : 10.1289/ehp.11537

N. Ben-jonathan, E. Hugo, and T. Brandebourg, Effects of bisphenol A on adipokine release from human adipose tissue: Implications for the metabolic syndrome, Molecular and Cellular Endocrinology, vol.304, issue.1-2, pp.49-54, 2009.
DOI : 10.1016/j.mce.2009.02.022

P. Alonso-magdalena, I. Quesada, and A. Nadal, Endocrine disruptors in the etiology of type 2 diabetes mellitus, Nature Reviews Endocrinology, vol.6, issue.6, pp.346-53, 2011.
DOI : 10.1038/nrendo.2011.56

M. Fernandez, J. Arrebola, and J. Taoufiki, Bisphenol-A and chlorinated derivatives in adipose tissue of women, Reproductive Toxicology, vol.24, issue.2, pp.259-64, 2007.
DOI : 10.1016/j.reprotox.2007.06.007

C. Philippat, M. Mortamais, and C. Chevrier, Exposure to Phthalates and Phenols during Pregnancy and Offspring Size at Birth, Environmental Health Perspectives, vol.120, issue.3, pp.464-70, 2012.
DOI : 10.1289/ehp.1103634

URL : https://hal.archives-ouvertes.fr/hal-00682887

J. Carwile and K. Michels, Urinary bisphenol A and obesity: NHANES 2003???2006, Environmental Research, vol.111, issue.6, pp.825-855, 2003.
DOI : 10.1016/j.envres.2011.05.014

T. Wang, M. Li, and B. Chen, Urinary Bisphenol A (BPA) Concentration Associates with Obesity and Insulin Resistance, The Journal of Clinical Endocrinology & Metabolism, vol.97, issue.2, pp.223-230, 2012.
DOI : 10.1210/jc.2011-1989

T. Takeuchi, O. Tsutsumi, and Y. Ikezuki, Positive Relationship between Androgen and the Endocrine Disruptor, Bisphenol A, in Normal Women and Women with Ovarian Dysfunction, Endocrine Journal, vol.51, issue.2, pp.165-174, 2004.
DOI : 10.1507/endocrj.51.165

E. Kandaraki, A. Chatzigeorgiou, and S. Livadas, Endocrine Disruptors and Polycystic Ovary Syndrome (PCOS): Elevated Serum Levels of Bisphenol A in Women with PCOS, The Journal of Clinical Endocrinology & Metabolism, vol.96, issue.3, pp.480-484, 2011.
DOI : 10.1210/jc.2010-1658

V. Bindhumol, K. Chitra, and P. Mathur, Bisphenol A induces reactive oxygen species generation in the liver of male rats, Toxicology, vol.188, issue.2-3, pp.117-141, 2003.
DOI : 10.1016/S0300-483X(03)00056-8

K. Sakurai, M. Kawazuma, and T. Adachi, Bisphenol A affects glucose transport in mouse 3T3-F442A adipocytes, British Journal of Pharmacology, vol.275, issue.Suppl 7, pp.209-223, 2004.
DOI : 10.1038/sj.bjp.0705520

P. Alonso-magdalena, O. Laribi, and A. Ropero, Low Doses of Bisphenol A and Diethylstilbestrol Impair Ca2+ Signals in Pancreatic ??-Cells through a Nonclassical Membrane Estrogen Receptor within Intact Islets of Langerhans, Environmental Health Perspectives, vol.113, issue.8, pp.969-77, 2005.
DOI : 10.1289/ehp.8002

A. Nadal, A. Ropero, and O. Laribi, Nongenomic actions of estrogens and xenoestrogens by binding at a plasma membrane receptor unrelated to estrogen receptor alpha and estrogen receptor beta, Proceedings of the National Academy of Sciences, vol.97, issue.21, pp.11603-11611, 2000.
DOI : 10.1073/pnas.97.21.11603

I. Quesada, E. Fuentes, and M. Viso-león, Low doses of the endocrine disruptor bisphenol A and the native hormone 17beta-estradiol rapidly activate transcription factor CREB, FASEB J, vol.16, pp.1671-1674, 2002.

S. Soriano, P. Alonso-magdalena, and M. García-arévalo, Rapid Insulinotropic Action of Low Doses of Bisphenol-A on Mouse and Human Islets of Langerhans: Role of Estrogen Receptor ??, PLoS ONE, vol.117, issue.2, p.31109, 2012.
DOI : 10.1371/journal.pone.0031109.s001

D. Melzer, N. Rice, and C. Lewis, Association of Urinary Bisphenol A Concentration with Heart Disease: Evidence from NHANES 2003/06, PLoS ONE, vol.5, issue.1, p.8673, 2003.
DOI : 10.1371/journal.pone.0008673.t005

M. Silver, O. Neill, M. Sowers, and M. , Urinary Bisphenol A and Type-2 Diabetes in U.S. Adults: Data from NHANES 2003-2008, PLoS ONE, vol.118, issue.10, p.26868, 2003.
DOI : 10.1371/journal.pone.0026868.t004

A. Shankar and S. Teppala, Relationship Between Body Mass Index and High Cystatin Levels Among US Adults, The Journal of Clinical Hypertension, vol.121, issue.suppl 4, pp.925-955, 2011.
DOI : 10.1111/j.1751-7176.2011.00548.x

M. Fernández, N. Bourguignon, and V. Lux-lantos, Neonatal Exposure to Bisphenol A and Reproductive and Endocrine Alterations Resembling the Polycystic Ovarian Syndrome in Adult Rats, Environmental Health Perspectives, vol.118, issue.9, pp.1217-1239, 2010.
DOI : 10.1289/ehp.0901257

W. Zhou, J. Liu, and L. Liao, Effect of bisphenol A on steroid hormone production in rat ovarian theca-interstitial and granulosa cells, Molecular and Cellular Endocrinology, vol.283, issue.1-2, pp.12-20, 2008.
DOI : 10.1016/j.mce.2007.10.010

N. Skakkebaeck, R. Meyts, E. Main, and K. , Testicular dysgenesis syndrome: an increasingly common developmental disorder with environmental aspects, Human Reproduction, vol.16, issue.5, pp.972-980, 2001.
DOI : 10.1093/humrep/16.5.972

K. Main, N. Skakkebaek, and J. Toppari, Cryptorchidism as Part of the Testicular Dysgenesis Syndrome: The Environmental Connection, Endocr Dev, vol.14, pp.167-73, 2009.
DOI : 10.1159/000207485

F. Brucker-davis, K. Wagner-mahler, and I. Delattre, Cryptorchidism at birth in Nice area (France) is associated with higher prenatal exposure to PCBs and DDE, as assessed by colostrum concentrations, Human Reproduction, vol.23, issue.8, pp.1708-1726, 2008.
DOI : 10.1093/humrep/den186

P. Fénichel, H. Déchaux, and C. Harthe, Unconjugated bisphenol A cord blood levels in boys with descended or undescended testes, Human Reproduction, vol.27, issue.4, pp.983-90, 2012.
DOI : 10.1093/humrep/der451

P. Fénichel, N. Lahlou, P. Coquillard, M. Pugeat, P. Pacini et al., Cord blood Insulin-like peptide 3 (INSL3) is reduced in idiopathic cryptorchidism: the missing link between fetal exposure to endocrine disruptors like bisphenol A and undescended testis? Oral communication presented at The Endocrine Society's Annual Meeting, 2013.

C. Richter, L. Birnbaum, and F. Farabollini, In vivo effects of bisphenol A in laboratory rodent studies, Reproductive Toxicology, vol.24, issue.2, pp.199-224, 2007.
DOI : 10.1016/j.reprotox.2007.06.004

D. Nakamura, Y. Yanagiba, and Z. Duan, Bisphenol A may cause testosterone reduction by adversely affecting both testis and pituitary systems similar to estradiol, Toxicology Letters, vol.194, issue.1-2, pp.16-25, 2010.
DOI : 10.1016/j.toxlet.2010.02.002

S. Salian, T. Doshi, and G. Vanage, Neonatal exposure of male rats to Bisphenol A impairs fertility and expression of sertoli cell junctional proteins in the testis, Toxicology, vol.265, issue.1-2, pp.56-67, 2009.
DOI : 10.1016/j.tox.2009.09.012

A. Bouskine, M. Nebout, and F. Brücker-davis, Low Doses of Bisphenol A Promote Human Seminoma Cell Proliferation by Activating PKA and PKG via a Membrane G-Protein???Coupled Estrogen Receptor, Environmental Health Perspectives, vol.117, issue.7, pp.1053-1061, 2009.
DOI : 10.1289/ehp.0800367

N. Chevalier, A. Bouskine, and P. Fenichel, Bisphenol A promotes testicular seminoma cell proliferation through GPER/GPR30, International Journal of Cancer, vol.146, issue.1, pp.241-243, 2012.
DOI : 10.1002/ijc.25972

S. Salian, T. Doshi, and G. Vanage, Perinatal exposure of rats to Bisphenol A affects the fertility of male offspring, Life Sciences, vol.85, issue.21-22, pp.742-52, 2009.
DOI : 10.1016/j.lfs.2009.10.004

D. Li, Z. Zhou, and M. Miao, Urine bisphenol-A (BPA) level in relation to semen quality, Fertility and Sterility, vol.95, issue.2, pp.625-655, 2011.
DOI : 10.1016/j.fertnstert.2010.09.026

P. Fénichel and F. Brucker-davis, Perturbateurs endocriniens environnementaux et cancer du sein??: de nouveaux facteurs de risque???, Gyn??cologie Obst??trique & Fertilit??, vol.36, issue.10, pp.969-77, 2008.
DOI : 10.1016/j.gyobfe.2008.05.006

C. Markey, E. Luque, M. De-toro, and M. , In Utero Exposure to Bisphenol A Alters the Development and Tissue Organization of the Mouse Mammary Gland1, Biology of Reproduction, vol.65, issue.4, pp.1215-1238, 2001.
DOI : 10.1093/biolreprod/65.4.1215

C. Markey, P. Wadi, and B. Rubin, Long-Term Effects of Fetal Exposure to Low Doses of the Xenoestrogen Bisphenol-A in the Female Mouse Genital Tract1, Biology of Reproduction, vol.72, issue.6, pp.1344-51, 2005.
DOI : 10.1095/biolreprod.104.036301

L. Vandenberg, M. Maffini, and P. Wadia, Exposure to Environmentally Relevant Doses of the Xenoestrogen Bisphenol-A Alters Development of the Fetal Mouse Mammary Gland, Endocrinology, vol.148, issue.1, pp.116-143, 2007.
DOI : 10.1210/en.2006-0561

M. Durando, L. Kass, and J. Piva, Prenatal Bisphenol A Exposure Induces Preneoplastic Lesions in the Mammary Gland in Wistar Rats, Environmental Health Perspectives, vol.115, issue.1, pp.80-86, 2007.
DOI : 10.1289/ehp.9282

J. Van-meeuwen, T. Burg, W. Piersma, and A. , Mixture effects of estrogenic compounds on proliferation and pS2 expression of MCF-7 human breast cancer cells, Food and Chemical Toxicology, vol.45, issue.11, pp.2319-2349, 2007.
DOI : 10.1016/j.fct.2007.06.011

E. Lapensee, T. Tuttle, and S. Fox, Bisphenol A at Low Nanomolar Doses Confers Chemoresistance in Estrogen Receptor-?????Positive and ???Negative Breast Cancer Cells, Environmental Health Perspectives, vol.117, issue.2, pp.175-80, 2009.
DOI : 10.1289/ehp.11788

J. Brody, K. Moysich, and O. Humblet, Environmental pollutants and breast cancer, Cancer, vol.147, issue.S12, pp.2667-711, 2007.
DOI : 10.1002/cncr.22655

X. Rébillard, B. Tretare, and A. Villers, The epidemiology of prostate cancer, Rev Prat, vol.53, pp.2224-2232, 2003.

G. Prins and K. Korach, The role of estrogens and estrogen receptors in normal prostate growth and disease, Steroids, vol.73, issue.3, pp.233-277, 2008.
DOI : 10.1016/j.steroids.2007.10.013

C. Yomenura, G. Cunha, and Y. Sugimura, Temporal and spatial factors in diethylstilbestrol-induced sqamous metaplasia in the developing human prostate. II. Persistent changes after removal of diethylstilbestrol, Acta Anat, vol.153, pp.1-11, 1995.

L. Multigner, J. Ndong, and A. Giusti, Chlordecone Exposure and Risk of Prostate Cancer, Journal of Clinical Oncology, vol.28, issue.21, pp.3457-62, 2010.
DOI : 10.1200/JCO.2009.27.2153

URL : https://hal.archives-ouvertes.fr/inserm-00518099

G. Prins, W. Tang, and J. Belmonte, Perinatal Exposure to Oestradiol and Bisphenol A Alters the Prostate Epigenome and Increases Susceptibility to Carcinogenesis, Basic & Clinical Pharmacology & Toxicology, vol.37, issue.2, pp.134-142, 2008.
DOI : 10.1111/j.1742-7843.2007.00166.x

M. Esteller, ABERRANT DNA METHYLATION AS A CANCER-INDUCING MECHANISM, Annual Review of Pharmacology and Toxicology, vol.45, issue.1, pp.629-56, 2005.
DOI : 10.1146/annurev.pharmtox.45.120403.095832

J. Hess-wilson, Bisphenol A may reduce the efficacy of androgen deprivation therapy in prostate cancer, Cancer Causes & Control, vol.117, issue.2, pp.1029-1066, 2009.
DOI : 10.1007/s10552-009-9337-8

F. Bray, L. Richiardi, and A. Ekbom, Trends in testicular cancer incidence and mortality in 22 European countries: Continuing increases in incidence and declines in mortality, International Journal of Cancer, vol.12, issue.20, pp.3099-111, 2006.
DOI : 10.1002/ijc.21747

R. Meyts and E. , Developmental model for the pathogenesis of testicular carcinoma in situ: genetic and environmental aspects, Human Reproduction Update, vol.12, issue.3, pp.303-326, 2006.
DOI : 10.1093/humupd/dmk006

P. Mills, G. Newell, and D. Johnson, TESTICULAR CANCER ASSOCIATED WITH EMPLOYMENT IN AGRICULTURE AND OIL AND NATURAL GAS EXTRACTION, The Lancet, vol.323, issue.8370, pp.207-217, 1984.
DOI : 10.1016/S0140-6736(84)92125-1

P. Mills, Correlation Analysis of Pesticide Use Data and Cancer Incidence Rates in California Counties, Archives of Environmental Health: An International Journal, vol.137, issue.6, pp.410-413, 1998.
DOI : 10.1080/00039899809605729

L. Hardell, B. Van-bavel, and G. Lindström, Increased Concentrations of Polychlorinated Biphenyls, Hexachlorobenzene, and Chlordanes in Mothers of Men with Testicular Cancer, Environmental Health Perspectives, vol.111, issue.7, pp.930-934, 2003.
DOI : 10.1289/ehp.5816

C. Roger, S. Lombard, and B. Mograbi, Estrogen-induced growth inhibition of human seminoma cells expressing estrogen receptor ?? and aromatase, Journal of Molecular Endocrinology, vol.35, issue.1, pp.191-200, 2005.
DOI : 10.1677/jme.1.01704

A. Bouskine, M. Nebout, and B. Mograbi, Estrogens Promote Human Testicular Germ Cell Cancer through a Membrane-Mediated Activation of Extracellular Regulated Kinase and Protein Kinase A, Endocrinology, vol.149, issue.2, pp.565-73, 2008.
DOI : 10.1210/en.2007-1318

N. Chevalier, A. Vega, and A. Bouskine, GPR30, the Non-Classical Membrane G Protein Related Estrogen Receptor, Is Overexpressed in Human Seminoma and Promotes Seminoma Cell Proliferation, PLoS ONE, vol.117, issue.4, p.34672, 2012.
DOI : 10.1371/journal.pone.0034672.s001

L. Vandenberg, I. Chahoud, and J. Heindel, Urinary, Circulating, and Tissue Biomonitoring Studies Indicate Widespread Exposure to Bisphenol A, Environmental Health Perspectives, vol.118, issue.8, pp.1055-70, 2010.
DOI : 10.1289/ehp.0901716

S. Ho, W. Tang, and J. Belmonte-de-frausto, Developmental Exposure to Estradiol and Bisphenol A Increases Susceptibility to Prostate Carcinogenesis and Epigenetically Regulates Phosphodiesterase Type 4 Variant 4, Cancer Research, vol.66, issue.11, pp.5624-5656, 2006.
DOI : 10.1158/0008-5472.CAN-06-0516

K. Moriyama, T. Tagami, and T. Akamizu, Thyroid Hormone Action Is Disrupted by Bisphenol A as an Antagonist, The Journal of Clinical Endocrinology & Metabolism, vol.87, issue.11, pp.5185-90, 2002.
DOI : 10.1210/jc.2002-020209

J. Chevrier, R. Gunier, and A. Bradman, Maternal urinary bisphenol A during pregnancy and maternal and neonatal thyroid function in the CHAMACOS study, Environ Health Perspect, vol.121, pp.138-182, 2013.

N. Shibata, J. Matsumoto, and K. Nakada, Male-specific suppression of hepatic microsomal UDP-glucuronosyl transferase activities toward sex hormones in the adult male rat administered bisphenol A, Biochemical Journal, vol.368, issue.3, pp.783-791, 2002.
DOI : 10.1042/bj20020804

F. Brucker-davis, P. Ferrari, M. Boda-buccino, K. Wagner-mahler, P. Pacini et al., Cord blood thyroid tests in boys born with and without cryptorchidism: correlations with birth parameters and in utero xenobiotics exposure, Thyroid, vol.10, pp.1133-1174, 2011.

W. Völkel, T. Colnot, and G. Csanády, Metabolism and Kinetics of Bisphenol A in Humans at Low Doses Following Oral Administration, Chemical Research in Toxicology, vol.15, issue.10, pp.1281-1288, 2002.
DOI : 10.1021/tx025548t

W. Dekant and W. Völkel, Human exposure to bisphenol A by biomonitoring: Methods, results and assessment of environmental exposures, Toxicology and Applied Pharmacology, vol.228, issue.1, pp.114-148, 2008.
DOI : 10.1016/j.taap.2007.12.008

G. Ginsberg and D. Rice, Does Rapid Metabolism Ensure Negligible Risk from Bisphenol A?, Environmental Health Perspectives, vol.117, issue.11, pp.1639-1682, 2009.
DOI : 10.1289/ehp.0901010

J. Matsumoto, H. Yokota, and A. Yuasa, Developmental Increases in Rat Hepatic Microsomal UDP-Glucuronosyltransferase Activities toward Xenoestrogens and Decreases during Pregnancy, Environmental Health Perspectives, vol.110, issue.2, pp.193-199, 2002.
DOI : 10.1289/ehp.02110193

M. Coughtrie, B. Burchell, and J. Leakey, The inadequacy of perinatal glucuronidation: immunoblot analysis of the developmental expression of individual UDP-glucuronosyl transferase isoenzymes in rat and human liver microsomes, Mol Pharmacol, vol.34, pp.729-764, 1988.

N. Kaddar, N. Bendridi, and C. Harthé, Development of a radioimmunoassay for the measurement of Bisphenol A in biological samples, Analytica Chimica Acta, vol.645, issue.1-2, pp.1-4, 2009.
DOI : 10.1016/j.aca.2009.04.036

C. Olsen, E. Meussen-elholm, and M. Samuelsen, Effects of the Environmental Oestrogens Bisphenol A, Tetrachlorobisphenol A, Tetrabromobisphenol A, 4-Hydroxybiphenyl and 4,4'-Dihydroxybiphenyl on Oestrogen Receptor Binding, Cell Proliferation and Regulation of Oestrogen Sensitive Proteins in the Human Breast Cancer Cell Line MCF-7, Pharmacology and Toxicology, vol.130, issue.4, pp.180-188, 2003.
DOI : 10.1021/tx000146b

R. Sharpe, Is It Time to End Concerns over the Estrogenic Effects of Bisphenol A?, Toxicological Sciences, vol.114, issue.1, pp.1-4, 2010.
DOI : 10.1093/toxsci/kfp299

H. Okada, T. Tokunaga, and X. Liu, Direct Evidence Revealing Structural Elements Essential for the High Binding Ability of Bisphenol A to Human Estrogen-Related Receptor-??, Environmental Health Perspectives, vol.116, issue.1, pp.32-40, 2008.
DOI : 10.1289/ehp.10587

V. Giguère, Transcriptional Control of Energy Homeostasis by the Estrogen-Related Receptors, Endocrine Reviews, vol.29, issue.6, pp.677-96, 2008.
DOI : 10.1210/er.2008-0017

M. Pupo, A. Pisano, and R. Lappano, Bisphenol A Induces Gene Expression Changes and Proliferative Effects through GPER in Breast Cancer Cells and Cancer-Associated Fibroblasts, Environmental Health Perspectives, vol.120, issue.8, pp.1177-82, 2012.
DOI : 10.1289/ehp.1104526

H. Sun, L. Xu, and J. Chen, Effect of bisphenol A, tetrachlorobisphenol A and pentachlorophenol on the transcriptional activities of androgen receptor-mediated reporter gene, Food and Chemical Toxicology, vol.44, issue.11, pp.1916-1937, 2006.
DOI : 10.1016/j.fct.2006.06.013

H. Wright, C. Clish, and T. Mikami, A Synthetic Antagonist for the Peroxisome Proliferator-activated Receptor ?? Inhibits Adipocyte Differentiation, Journal of Biological Chemistry, vol.275, issue.3, pp.1873-1880, 2000.
DOI : 10.1074/jbc.275.3.1873

R. Zoeller, R. Bansal, C. Parris, and A. Bisphenol, , Increases Serum Thyroxine, and Alters RC3/Neurogranin Expression in the Developing Rat Brain, Endocrinology, vol.146, issue.2, pp.607-619, 2005.
DOI : 10.1210/en.2004-1018

J. Bromer, J. Wu, and Y. Zhou, Diethylstilbestrol Exposure: An Epigenetic Mechanism for Altered Developmental Programming, Endocrinology, vol.150, issue.7, pp.3376-82, 2009.
DOI : 10.1210/en.2009-0071

S. Li, R. Hansman, and R. Newbold, Neonatal diethylstilbestrol exposure induces persistent elevation of c-fos expression and hypomethylation in its exon-4 in mouse uterus, Molecular Carcinogenesis, vol.4, issue.2, pp.78-84, 2003.
DOI : 10.1002/mc.10147

J. Bromer, Y. Zhou, and M. Taylor, Bisphenol-A exposure in utero leads to epigenetic alterations in the developmental programming of uterine estrogen response, The FASEB Journal, vol.24, issue.7, pp.2273-80, 2010.
DOI : 10.1096/fj.09-140533

X. Fei, H. Chung, and H. Taylor, Methoxychlor Disrupts Uterine Hoxa10 Gene Expression, Endocrinology, vol.146, issue.8, pp.3445-51, 2005.
DOI : 10.1210/en.2005-0341

URL : http://citeseerx.ist.psu.edu/viewdoc/summary?doi=

M. Esteller and K. Aberrant, ABERRANT DNA METHYLATION AS A CANCER-INDUCING MECHANISM, Annual Review of Pharmacology and Toxicology, vol.45, issue.1, pp.629-56, 2005.
DOI : 10.1146/annurev.pharmtox.45.120403.095832

W. Tang, R. Newbold, and M. Mardilovitch, 1 in Mouse Uteri Neonatally Exposed to Diethylstilbestrol or Genistein, Endocrinology, vol.149, issue.12, pp.5922-5961, 2008.
DOI : 10.1210/en.2008-0682

D. Volle, M. Decourteix, and E. Garo, The orphan nuclear receptor small heterodimer partner mediates male infertility induced by diethylstilbestrol in mice, Journal of Clinical Investigation, vol.119, issue.12, pp.3752-64, 2009.
DOI : 10.1172/JCI38521DS1

K. Chiam, W. Tilley, and L. Butler, The dynamic and static modification of the epigenome by hormones: A role in the developmental origin of hormone related cancers, Biochimica et Biophysica Acta (BBA) - Reviews on Cancer, vol.1795, issue.2, pp.104-113, 2009.
DOI : 10.1016/j.bbcan.2008.12.001