Effect of inhibition of the mitochondrial ATPase on net myocardial ATP in total ischemia, Journal of Molecular and Cellular Cardiology, vol.23, issue.12, pp.1383-1395, 1991. ,
DOI : 10.1016/0022-2828(91)90185-O
Declines in mitochondrial respiration during cardiac reperfusion: Age-dependent inactivation of ??-ketoglutarate dehydrogenase, Proceedings of the National Academy of Sciences, vol.96, issue.12, pp.6689-6693, 1999. ,
DOI : 10.1073/pnas.96.12.6689
Selective inactivation of redox-sensitive mitochondrial enzymes during cardiac reperfusion, Archives of Biochemistry and Biophysics, vol.406, issue.2, pp.222-228, 2002. ,
DOI : 10.1016/S0003-9861(02)00446-0
The differential effects of superoxide anion, hydrogen peroxide and hydroxyl radical on cardiac mitochondrial oxidative phosphorylation, Free Radical Research, vol.108, issue.10 ,
DOI : 10.1074/jbc.M208262200
Decreased complex III activity in mitochondria isolated from rat heart subjected to ischemia and reperfusion: role of reactive oxygen species and cardiolipin, The FASEB Journal, vol.17, pp.714-716, 2003. ,
DOI : 10.1096/fj.02-0729fje
Decrease in Mitochondrial Complex I Activity in Ischemic/Reperfused Rat Heart: Involvement of Reactive Oxygen Species and Cardiolipin, Circulation Research, vol.94, issue.1, pp.53-59, 2004. ,
DOI : 10.1161/01.RES.0000109416.56608.64
-dependent pore activated by oxidative stress in heart mitochondria, Biochemical Journal, vol.245, issue.3, pp.915-918, 1987. ,
DOI : 10.1042/bj2450915
URL : https://hal.archives-ouvertes.fr/hal-01159417
Mitochondrial non-specific pores remain closed during cardiac ischaemia, but open upon reperfusion, Biochemical Journal, vol.307, issue.1, pp.93-98, 1995. ,
DOI : 10.1042/bj3070093
Inhibition of mitochondrial permeability transition prevents mitochondrial dysfunction, cytochrome c release and apoptosis induced by heart ischemia, Journal of Molecular and Cellular Cardiology, vol.35, issue.4, pp.357-366, 2003. ,
DOI : 10.1016/S0022-2828(03)00005-1
The Pathophysiology of Mitochondrial Cell Death, Science, vol.305, issue.5684, pp.626-629, 2004. ,
DOI : 10.1126/science.1099320
A tale of two mitochondrial channels, MAC and PTP, in apoptosis, Apoptosis, vol.23, issue.Suppl 1, pp.857-868, 2007. ,
DOI : 10.1007/s10495-007-0722-z
Heart mitochondria: gates of life and death, Cardiovascular Research, vol.77, issue.2, pp.334-343, 2008. ,
DOI : 10.1093/cvr/cvm005
Mitochondrial membrane permeability transition and cell death, Biochimica et Biophysica Acta (BBA) - Bioenergetics, vol.1757, issue.9-10, pp.1297-1300, 2006. ,
DOI : 10.1016/j.bbabio.2006.03.017
Loss of cyclophilin D reveals a critical role for mitochondrial permeability transition in cell death, Nature, vol.65, issue.7033, pp.658-662, 2005. ,
DOI : 10.1038/sj.gt.3301048
Cyclophilin D-dependent mitochondrial permeability transition regulates some necrotic but not apoptotic cell death, Nature, vol.12, issue.7033, pp.652-658, 2005. ,
DOI : 10.1084/jem.189.11.1699
Cyclophilin D is a component of mitochondrial permeability transition and mediates neuronal cell death after focal cerebral ischemia, Proceedings of the National Academy of Sciences, vol.102, issue.34, pp.12005-12010, 2005. ,
DOI : 10.1073/pnas.0505294102
The ADP/ATP translocator is not essential for the mitochondrial permeability transition pore, Nature, vol.427, issue.6973, pp.461-465, 2004. ,
DOI : 10.1038/nature02229
The cyclophilin-D binding protein of the mitochondrial permeability transition pore may not be the adenine nucleotide translocase, Journal of Molecular and Cellular Cardiology, vol.42, issue.6, p.117, 2007. ,
DOI : 10.1016/j.yjmcc.2007.03.269
The Identity and Regulation of the Mitochondrial Permeability Transition Pore, Annals of the New York Academy of Sciences, vol.2, issue.1, pp.197-212, 2008. ,
DOI : 10.1196/annals.1420.023
Voltage-dependent anion channels are dispensable for mitochondrial-dependent cell death, Nature Cell Biology, vol.336, issue.5, pp.550-555, 2007. ,
DOI : 10.1074/jbc.M313717200
Apoptotic Pathways: Ten Minutes to Dead, Cell, vol.121, issue.5, pp.671-674, 2005. ,
DOI : 10.1016/j.cell.2005.05.019
THE MITOCHONDRIAL DEATH/LIFE REGULATOR IN APOPTOSIS AND NECROSIS, Annual Review of Physiology, vol.60, issue.1, pp.619-642, 1998. ,
DOI : 10.1146/annurev.physiol.60.1.619
Bcl-2 family proteins and mitochondria, Biochimica et Biophysica Acta (BBA) - Bioenergetics, vol.1366, issue.1-2, pp.127-137, 1998. ,
DOI : 10.1016/S0005-2728(98)00108-X
Mitochondria as the central control point of apoptosis, Trends in Cell Biology, vol.10, issue.9, pp.369-377, 2000. ,
DOI : 10.1016/S0962-8924(00)01803-1
Inhibition of anoxia-induced injury in heart myocytes by cyclosporin A, Journal of Molecular and Cellular Cardiology, vol.23, issue.12, pp.1351-1354, 1991. ,
DOI : 10.1016/0022-2828(91)90181-K
Calcium preconditioning inhibits mitochondrial permeability transition and apoptosis, Am J Physiol Heart Circ Physiol, vol.280, pp.899-908, 2001. ,
Protection by Cyclosporin A of Ischemia/Reperfusion-Induced Damage in Isolated Rat Hearts, Journal of Molecular and Cellular Cardiology, vol.25, issue.12, pp.1461-1469, 1993. ,
DOI : 10.1006/jmcc.1993.1162
Preconditioning delays Ca2+-induced mitochondrial permeability transition, Cardiovascular Research, vol.61, issue.1, pp.115-122, 2004. ,
DOI : 10.1016/j.cardiores.2003.11.003
Mitochondrial permeability transition pore as a target for cardioprotection in the human heart, AJP: Heart and Circulatory Physiology, vol.289, issue.1, pp.237-242, 2005. ,
DOI : 10.1152/ajpheart.01192.2004
Mitochondria as target for antiischemic drugs, Advanced Drug Delivery Reviews, vol.49, issue.1-2, pp.151-174, 2001. ,
DOI : 10.1016/S0169-409X(01)00132-6
Mitochondrial permeability transition pore opening during myocardial reperfusion???a target for cardioprotection, Cardiovascular Research, vol.61, issue.3, pp.372-385, 2004. ,
DOI : 10.1016/S0008-6363(03)00533-9
Effect of Cyclosporine on Reperfusion Injury in Acute Myocardial Infarction, New England Journal of Medicine, vol.359, issue.5 ,
DOI : 10.1056/NEJMoa071142
URL : https://hal.archives-ouvertes.fr/hal-00428144
Postconditioning Inhibits Mitochondrial Permeability Transition, Circulation, vol.111, issue.2, pp.194-197, 2005. ,
DOI : 10.1161/01.CIR.0000151290.04952.3B
Inhibition of mitochondrial permeability transition improves functional recovery and reduces mortality following acute myocardial infarction in mice, AJP: Heart and Circulatory Physiology, vol.293, issue.3, pp.1654-1661, 2007. ,
DOI : 10.1152/ajpheart.01378.2006
Sanglifehrin A Acts as a Potent Inhibitor of the Mitochondrial Permeability Transition and Reperfusion Injury of the Heart by Binding to Cyclophilin-D at a Different Site from Cyclosporin A, Journal of Biological Chemistry, vol.277, issue.38, pp.34793-34799, 2002. ,
DOI : 10.1074/jbc.M202191200
Inhibiting mitochondrial permeability transition pore opening at reperfusion protects against ischaemia???reperfusion injury, Cardiovascular Research, vol.60, issue.3 ,
DOI : 10.1016/j.cardiores.2003.09.025
URL : http://cardiovascres.oxfordjournals.org/cgi/content/short/60/3/617
Mitochondrial permeability transition-pore inhibition enhances functional recovery after long-time hypothermic heart preservation, Transplantation, vol.76, issue.9, pp.1314-1320, 2003. ,
DOI : 10.1097/01.TP.0000085660.93090.79
Evidence for the existence of 3H -trimetazidine binding sites involved in the regulation ,
Clinically Approved Heterocyclics Act on a Mitochondrial Target and Reduce Stroke-induced Pathology, The Journal of Experimental Medicine, vol.55, issue.2, pp.211-222, 2004. ,
DOI : 10.1016/S0014-2999(03)01303-7
Inhibition of the mitochondrial permeability transition by cyclosporin A during long time frame experiments: relationship between pore opening and the activity of mitochondrial phospholipases, Biochemistry, vol.34, issue.50, pp.16440-16449, 1995. ,
DOI : 10.1021/bi00050a027
A Ubiquinone-binding Site Regulates the Mitochondrial Permeability Transition Pore, Journal of Biological Chemistry, vol.273, issue.40, pp.25734-25734, 1998. ,
DOI : 10.1074/jbc.273.40.25734
The Voltage-dependent Anion Channel Is the Target for a New Class of Inhibitors of the Mitochondrial Permeability Transition Pore, Journal of Biological Chemistry, vol.278, issue.50, pp.49812-49818, 2003. ,
DOI : 10.1074/jbc.M304748200
Properties of the permeability transition in VDAC1???/??? mitochondria, Biochimica et Biophysica Acta (BBA) - Bioenergetics, vol.1757, issue.5-6, pp.590-595, 2006. ,
DOI : 10.1016/j.bbabio.2006.02.007
Regulation of the mitochondrial permeability transition pore by ubiquinone analogs. A progress report mitochondrial membrane permeability, a pharmacological target for cardioprotection, Free Radic Res . Curr Med Chem . Author manuscript Page, vol.36, issue.11, pp.405-412, 2002. ,
URL : https://hal.archives-ouvertes.fr/inserm-00389972
in cardiovascular disease, BioFactors, vol.120, issue.2, pp.273-284, 1999. ,
DOI : 10.1002/biof.5520090224
The Coenzyme Q10 Analog Decylubiquinone Inhibits the Redox-activated Mitochondrial Permeability Transition: ROLE OF MITOCHONDRIAL RESPIRATORY COMPLEX III, Journal of Biological Chemistry, vol.278, issue.49, pp.49079-49084, 2003. ,
DOI : 10.1074/jbc.M307841200
In vitro properties of 5-(benzylsulfonyl)-4-bromo-2-methyl-3(2H)-pyridazinone: A novel permeability transition pore inhibitor, European Journal of Pharmacology, vol.519, issue.1-2, pp.24-30, 2005. ,
DOI : 10.1016/j.ejphar.2005.06.046
Connected to Death: The (Unexpurgated) Mitochondrial Pathway of Apoptosis, Science, vol.310, issue.5745, pp.66-67, 2005. ,
DOI : 10.1126/science.1117105
Mitochondrial cytochrome c release in apoptosis occurs upstream of DEVD-specific caspase activation and independently of mitochondrial transmembrane depolarization, The EMBO Journal, vol.17, issue.1, pp.37-49, 1998. ,
DOI : 10.1093/emboj/17.1.37
Dissociation of Cytochrome c from the Inner Mitochondrial Membrane during Cardiac Ischemia, Journal of Biological Chemistry, vol.278, issue.36, pp.34499-34504, 2003. ,
DOI : 10.1074/jbc.M302021200
Role of the permeability transition pore in cytochrome C release from mitochondria during ischemia-reperfusion in rat liver, Biochemical Pharmacology, vol.68, issue.10 ,
DOI : 10.1016/j.bcp.2004.07.006
Preconditioning prevents loss in mitochondrial function and release of cytochrome c during prolonged cardiac ischemia/reperfusion, Archives of Biochemistry and Biophysics, vol.453, issue.1, pp.130-134, 2006. ,
DOI : 10.1016/j.abb.2006.02.007
Bcl-2 family members and apoptosis, taken to heart, AJP: Cell Physiology, vol.292, issue.1, pp.45-51, 2007. ,
DOI : 10.1152/ajpcell.00229.2006
Mitochondrio-nuclear translocation of AIF in apoptosis and necrosis, FASEB J, vol.14, pp.729-739, 2000. ,
URL : https://hal.archives-ouvertes.fr/hal-00315088
Endonuclease G is an apoptotic DNase when released from mitochondria, Nature, vol.412, issue.6842, pp.95-99, 2001. ,
DOI : 10.1038/35083620
The Mitochondrial Voltage-dependent Anion Channel (VDAC) as a Therapeutic Target for Initiating Cell Death, Current Medicinal Chemistry, vol.10, issue.16, pp.1527-1533, 2003. ,
DOI : 10.2174/0929867033457214
Distinct BH3 domains either sensitize or activate mitochondrial apoptosis, serving as prototype cancer therapeutics, Cancer Cell, vol.2, issue.3, pp.183-192, 2002. ,
DOI : 10.1016/S1535-6108(02)00127-7
Cardiac functional improvement by a human Bcl-2 transgene in a mouse model of ischemia/reperfusion injury, The Journal of Gene Medicine, vol.269, issue.5, pp.326-333, 2000. ,
DOI : 10.1002/1521-2254(200009/10)2:5<326::AID-JGM133>3.0.CO;2-1
An Essential Role of the Antioxidant Gene Bcl-2 in Myocardial Adaptation to Ischemia: An Insight with Antisense Bcl-2 Therapy, Antioxidants & Redox Signaling, vol.3, issue.3, pp.403-413, 2001. ,
DOI : 10.1089/15230860152409059
Overexpression of Bcl-2 attenuates apoptosis and protects against myocardial I/R injury in transgenic mice, Am J Physiol Heart Circ Physiol, vol.280, pp.2313-2320, 2001. ,
Bax ablation protects against myocardial ischemia-reperfusion injury in transgenic mice, American Journal of Physiology - Heart and Circulatory Physiology, vol.284, issue.6, pp.2351-2359, 2003. ,
DOI : 10.1152/ajpheart.00783.2002
Apoptosis Repressor with Caspase Recruitment Domain Protects against Cell Death by Interfering with Bax Activation, Journal of Biological Chemistry, vol.279, issue.20, pp.21233-21238, 2004. ,
DOI : 10.1074/jbc.M400695200
Bcl-xL Gene Transfer Inhibits Bax Translocation and Prolongs Cardiac Cold Preservation Time in Rats, Circulation, vol.112, issue.1, pp.76-83, 2005. ,
DOI : 10.1161/CIRCULATIONAHA.105.535740
Apoptosis-regulating proteins as targets for drug discovery, Trends in Molecular Medicine, vol.7, issue.7, pp.314-319, 2001. ,
DOI : 10.1016/S1471-4914(01)02026-3
Inhibition of Bax-induced cytochrome c release from neural cell and brain mitochondria by dibucaine and propranolol, J Neurosci, vol.23, pp.2735-2743, 2003. ,
6-dibromocarbazole piperazine derivatives of 2-propanol as first inhibitors of cytochrome c release via Bax channel modulation, J Med Chem, vol.3, issue.46, pp.4365-4368, 2003. ,
Cytoprotective membrane-permeable peptides designed from the Bax-binding domain of Ku70, Nature Cell Biology, vol.5, issue.4, pp.352-357, 2003. ,
DOI : 10.1038/ncb955
Bax Channel Inhibitors Prevent Mitochondrion-mediated Apoptosis and Protect Neurons in a Model of Global Brain Ischemia, Journal of Biological Chemistry, vol.280, issue.52, pp.42960-42970, 2005. ,
DOI : 10.1074/jbc.M505843200
The roles of Bid, APOPTOSIS, vol.7, issue.5, pp.433-440, 2002. ,
DOI : 10.1023/A:1020035124855
Bid Is Cleaved by Calpain to an Active Fragment in Vitro and during Myocardial Ischemia/Reperfusion, Journal of Biological Chemistry, vol.276, issue.33 ,
DOI : 10.1074/jbc.M103701200
Calpain and Mitochondria in Ischemia/Reperfusion Injury, Journal of Biological Chemistry, vol.277, issue.32, pp.29181-29186, 2002. ,
DOI : 10.1074/jbc.M204951200
Possible involvement of calpain activation in pathogenesis of chronic heart failure after acute myocardial infarction, J Cardiovasc Pharmacol, vol.47, pp.413-421, 2006. ,
The therapeutic potential of the calpain family: new aspects, Drug Discovery Today, vol.11, issue.19-20, pp.917-923, 2006. ,
DOI : 10.1016/j.drudis.2006.08.009
Structure-activity relationships by interligand NOE-based design and synthesis of antiapoptotic compounds targeting Bid, Proceedings of the National Academy of Sciences, vol.103, issue.33, pp.12602-12606, 2006. ,
DOI : 10.1073/pnas.0603460103
Redox Stress and the Contributions of BH3-Only Proteins to Infarction, Antioxidants & Redox Signaling, vol.8, issue.9-10 ,
DOI : 10.1089/ars.2006.8.1667
Inducible Expression of BNIP3 Provokes Mitochondrial Defects and Hypoxia-Mediated Cell Death of Ventricular Myocytes, Circulation Research, vol.91, issue.3, pp.226-231, 2002. ,
DOI : 10.1161/01.RES.0000029232.42227.16
Hypoxia and acidosis activate cardiac myocyte death through the Bcl-2 family protein BNIP3, Proceedings of the National Academy of Sciences, vol.99, issue.20, pp.12825-12830, 2002. ,
DOI : 10.1073/pnas.202474099
Bnip3 mediates mitochondrial dysfunction and cell death through Bax and Bak, Biochemical Journal, vol.405, issue.3, pp.407-415, 2007. ,
DOI : 10.1042/BJ20070319
Response to myocardial ischemia/reperfusion injury involves Bnip3 and autophagy, Cell Death and Differentiation, vol.185, issue.1, pp.146-157, 2007. ,
DOI : 10.1038/sj.cdd.4401936
Targeted deletion of Puma attenuates cardiomyocyte death and improves cardiac function during ischemia-reperfusion, AJP: Heart and Circulatory Physiology, vol.291, issue.1, pp.52-60, 2006. ,
DOI : 10.1152/ajpheart.01046.2005
VDAC: The channel at the interface between mitochondria and the cytosol, Molecular and Cellular Biochemistry, vol.256, issue.1/2, pp.107-115, 2004. ,
DOI : 10.1023/B:MCBI.0000009862.17396.8d
Characterization of rat porin isoforms: cloning of a cardiac type-3 variant encoding an additional methionine at its putative N-terminal region, Biochimica et Biophysica Acta (BBA) - Gene Structure and Expression, vol.1399, issue.1, pp.47-50, 1998. ,
DOI : 10.1016/S0167-4781(98)00088-8
All three isoforms of the voltage-dependent anion channel (VDAC1, VDAC2, and VDAC3) are present in mitochondria from bovine, rabbit, and rat brain, Arch Biochem Biophys, vol.422, pp.191-196, 2004. ,
Bcl-2 family proteins regulate the release of apoptogenic cytochrome c by the mitochondrial channel VDAC, Nature, vol.399, pp.483-487, 1999. ,
VDAC2 Inhibits BAK Activation and Mitochondrial Apoptosis, Science, vol.301, issue.5632, pp.513-517, 2003. ,
DOI : 10.1126/science.1083995
Bcl-xL Prevents Cell Death following Growth Factor Withdrawal by Facilitating Mitochondrial ATP/ADP Exchange, Molecular Cell, vol.3, issue.2, pp.159-167, 1999. ,
DOI : 10.1016/S1097-2765(00)80307-X
VDAC-dependent permeabilization of the outer mitochondrial membrane by superoxide induces rapid and massive cytochrome c release, J Cell Biol . Am J Physiol Heart Circ Physiol, vol.155, issue.280, pp.1003-1015, 2001. ,
channel opener diazoxide protects cardiac myocytes during metabolic inhibition without causing mitochondrial depolarization or flavoprotein oxidation, British Journal of Pharmacology, vol.28, issue.3, pp.535-542, 2001. ,
DOI : 10.1038/sj.bjp.0704289
Myocardial KATP Channels in Preconditioning, Circulation Research, vol.87, issue.10, pp.845-855, 2000. ,
DOI : 10.1161/01.RES.87.10.845
Transient Mitochondrial Permeability Transition Pore Opening Mediates Preconditioning-Induced Protection, Circulation, vol.109, issue.14, pp.1714-1717, 2004. ,
DOI : 10.1161/01.CIR.0000126294.81407.7D
URL : http://discovery.ucl.ac.uk/7775/1/7775.pdf
Does the Mitochondrial Permeability Transition Have a Role in Preconditioning? * Response, Circulation, vol.110, issue.11, p.303, 2004. ,
DOI : 10.1161/01.CIR.0000141458.28925.D2
The Mechanism by Which the Mitochondrial ATP-sensitive K+ Channel Opening and H2O2 Inhibit the Mitochondrial Permeability Transition, Journal of Biological Chemistry, vol.281, issue.30, pp.20801-20808, 2006. ,
DOI : 10.1074/jbc.M600959200
Integration of mitochondrial function, pp.257-276, 1998. ,
channel protects the ischemic heart, American Journal of Physiology - Heart and Circulatory Physiology, vol.283, issue.1, pp.284-295, 2002. ,
DOI : 10.1152/ajpheart.00034.2002
Mitochondrial potassium transport: the role of the mitochondrial ATP-sensitive K+ channel in cardiac function and cardioprotection, Biochimica et Biophysica Acta (BBA) - Bioenergetics, vol.1606, issue.1-3, pp.1-21, 2003. ,
DOI : 10.1016/S0005-2728(03)00109-9
Nicorandil improves cardiac function and clinical outcome in patients with acute myocardial infarction undergoing primary percutaneous coronary intervention: Role of inhibitory effect on reactive oxygen species formation, American Heart Journal, vol.148, issue.4, p.15, 2004. ,
DOI : 10.1016/j.ahj.2004.05.014
Impact of a Single Intravenous Administration of Nicorandil Before Reperfusion in Patients With ST-Segment-Elevation Myocardial Infarction, Circulation, vol.112, issue.9, pp.1284-1288, 2005. ,
DOI : 10.1161/CIRCULATIONAHA.104.530329
Human atrial natriuretic peptide and nicorandil as adjuncts to reperfusion treatment for acute myocardial infarction (J-WIND): two randomised trials, The Lancet, vol.370, issue.9597, pp.1483-14936, 2007. ,
DOI : 10.1016/S0140-6736(07)61634-1
The role of mitochondria in protection of the heart by preconditioning, Biochimica et Biophysica Acta (BBA) - Bioenergetics, vol.1767, issue.8, pp.1007-1031, 2007. ,
DOI : 10.1016/j.bbabio.2007.05.008
Translocator protein (18kDa): new nomenclature for the peripheral-type benzodiazepine receptor based on its structure and molecular function, Trends in Pharmacological Sciences, vol.27, issue.8, pp.402-409, 2006. ,
DOI : 10.1016/j.tips.2006.06.005
The mitochondrial benzodiazepine receptor: Evidence for association with the voltage-dependent anion channel (VDAC), Journal of Bioenergetics and Biomembranes, vol.38, issue.1, pp.63-69, 1992. ,
DOI : 10.1007/BF00769532
PK11195, a Ligand of the Mitochondrial Benzodiazepine Receptor, Facilitates the Induction of Apoptosis and Reverses Bcl-2-Mediated Cytoprotection, Experimental Cell Research, vol.241, issue.2, pp.426-434, 1998. ,
DOI : 10.1006/excr.1998.4084
Peripheral-type benzodiazepine receptor: structure and function of a cholesterol-binding protein in steroid and bile acid biosynthesis, Steroids, vol.68, issue.7-8 ,
DOI : 10.1016/S0039-128X(03)00101-6
Peripheral Benzodiazepine Receptor Agonists Exhibit Potent Antiapoptotic Activities, Biochemical and Biophysical Research Communications, vol.265, issue.2, pp.457-461, 1999. ,
DOI : 10.1006/bbrc.1999.1683
nvolvement of peripheral benzodiazepine receptors in the protection of hematopoietic cells against oxygen radical damage, Blood, vol.87, pp.3170-3178, 1996. ,
odulation of peripheral-type benzodiazepine receptor levels in a reperfusion injury pig kidney-graft model mitochondrial membrane permeability, a pharmacological target for cardioprotection, Transplantation . Curr Med Chem . Author manuscript Page, vol.74, issue.13, pp.1507-1515, 2002. ,
Role of Peripheral Benzodiazepine Receptors in Mitochondrial, Cellular, and Cardiac Damage Induced by Oxidative Stress and Ischemia-Reperfusion, Journal of Pharmacology and Experimental Therapeutics, vol.306, issue.3, pp.828-837, 2003. ,
DOI : 10.1124/jpet.103.052068
Peripheral Benzodiazepine Receptor-Induced Myocardial Protection is Mediated by Inhibition of Mitochondrial Membrane Permeabilization, Journal of Pharmacology and Experimental Therapeutics, vol.323, issue.1, pp.336-345, 2007. ,
DOI : 10.1124/jpet.107.124255
Preconditioning with ischemia: a delay of lethal cell injury in ischemic myocardium, Circulation, vol.74, issue.5, pp.1124-1136, 1986. ,
DOI : 10.1161/01.CIR.74.5.1124
New directions for protecting the heart against ischaemia???reperfusion injury: targeting the Reperfusion Injury Salvage Kinase (RISK)-pathway, Cardiovascular Research, vol.61, issue.3, pp.448-460, 2004. ,
DOI : 10.1016/j.cardiores.2003.09.024
Inhibition of myocardial injury by ischemic postconditioning during reperfusion: comparison with ischemic preconditioning, American Journal of Physiology - Heart and Circulatory Physiology, vol.285, issue.2, pp.579-588, 2003. ,
DOI : 10.1152/ajpheart.01064.2002
Preconditioning and postconditioning: United at reperfusion, Pharmacology & Therapeutics, vol.116, issue.2, pp.173-191, 2007. ,
DOI : 10.1016/j.pharmthera.2007.06.005
Long-Term Benefit of Postconditioning, Circulation, vol.117, issue.8, pp.1037-1044, 2008. ,
DOI : 10.1161/CIRCULATIONAHA.107.729780
URL : https://hal.archives-ouvertes.fr/hal-00428123
Glycogen synthase kinase-3?? mediates convergence of protection signaling to inhibit the mitochondrial permeability transition pore, Journal of Clinical Investigation, vol.113, issue.11, pp.1535-1549, 2004. ,
DOI : 10.1172/JCI19906DS1
Signalling via the reperfusion injury signalling kinase (RISK) pathway links closure of the mitochondrial permeability transition pore to cardioprotection, The International Journal of Biochemistry & Cell Biology, vol.38, issue.3, pp.414-419, 2006. ,
DOI : 10.1016/j.biocel.2005.09.017
PI 3-kinase regulates the mitochondrial transition pore in controlled reperfusion and postconditioning, Cardiovascular Research, vol.69, issue.1, pp.178-185, 2006. ,
DOI : 10.1016/j.cardiores.2005.07.014
Cardioprotective Effect of Morphine and a Blocker of Glycogen Synthase Kinase 3??, SB216763 [3-(2,4-Dichlorophenyl)-4(1-methyl-1H-indol-3-yl)-1H-pyrrole-2,5-dione], via Inhibition of the Mitochondrial Permeability Transition Pore, Journal of Pharmacology and Experimental Therapeutics, vol.326, issue.1, pp.252-258, 2008. ,
DOI : 10.1124/jpet.108.138008
Mitochondria as a Target for the Cardioprotective Effects of Nitric Oxide in Ischemia???Reperfusion Injury, Antioxidants & Redox Signaling, vol.10, issue.3, pp.579-599, 2008. ,
DOI : 10.1089/ars.2007.1845
Protein Kinase Cepsilon Interacts With and Inhibits the Permeability Transition Pore in Cardiac Mitochondria, Circulation Research, vol.92, issue.8, pp.873-880, 2003. ,
DOI : 10.1161/01.RES.0000069215.36389.8D
Role of the Mitochondrial Permeability Transition in Myocardial Disease, Circulation Research, vol.93, issue.4, pp.292-301, 2003. ,
DOI : 10.1161/01.RES.0000087542.26971.D4
Protein Kinase C?? Activation Induces Apoptosis in Response to Cardiac Ischemia and Reperfusion Damage: A MECHANISM INVOLVING BAD AND THE MITOCHONDRIA, Journal of Biological Chemistry, vol.279, issue.46, pp.47985-47991, 2004. ,
DOI : 10.1074/jbc.M405071200
Modulation of the mitochondrial permeability transition pore complex in GSK-3??-mediated myocardial protection, Journal of Molecular and Cellular Cardiology, vol.43, issue.5, pp.564-570, 2007. ,
DOI : 10.1016/j.yjmcc.2007.08.010
The Phosphatidylinositol 3-Kinase (PI3K)-Akt Pathway Suppresses Bax Translocation to Mitochondria, Journal of Biological Chemistry, vol.277, issue.16, pp.14040-14047, 2002. ,
DOI : 10.1074/jbc.M108975200
Ischemic preconditioning, insulin, and morphine all cause hexokinase redistribution, AJP: Heart and Circulatory Physiology, vol.289, issue.1, pp.496-499, 2005. ,
DOI : 10.1152/ajpheart.01182.2004
Brief repetitive balloon occlusions enhance reperfusion during percutaneous coronary intervention for acute myocardial infarction: A pilot study, Catheterization and Cardiovascular Interventions, vol.39, issue.3, pp.361-367, 2005. ,
DOI : 10.1002/ccd.20397
Reduction in myocardial infarct size by postconditioning in patients after percutaneous coronary intervention, J Invasive Cardiol, vol.19, pp.424-430, 2007. ,
Beneficial Effects of Intracoronary Adenosine as an Adjunct to Primary Angioplasty in Acute Myocardial Infarction, Circulation, vol.101, issue.18, pp.2154-2159, 2000. ,
DOI : 10.1161/01.CIR.101.18.2154
Left ventricular function and cardiovascular events following adjuvant therapy with adenosine in acute myocardial infarction treated with thrombolysis, European Journal of Clinical Pharmacology, vol.13, issue.1, pp.1-9, 2003. ,
DOI : 10.1016/S0735-1097(98)00604-4
Adenosine as an adjunct to thrombolytic therapy for acute myocardial infarction, Journal of the American College of Cardiology, vol.34, issue.6, pp.1711-1720, 1999. ,
DOI : 10.1016/S0735-1097(99)00418-0
A Randomized, Double-Blinded, Placebo-Controlled Multicenter Trial of Adenosine as an Adjunct to Reperfusion in the Treatment of Acute Myocardial Infarction (AMISTAD-II), Journal of the American College of Cardiology, vol.45, issue.11, pp.1775-1780, 2005. ,
DOI : 10.1016/j.jacc.2005.02.061
Opioid-Induced Cardioprotection Occurs via Glycogen Synthase Kinase ?? Inhibition During Reperfusion in Intact Rat Hearts, Circulation Research, vol.94, issue.7, pp.960-966, 2004. ,
DOI : 10.1161/01.RES.0000122392.33172.09
Inhibition of GSK3?? by Postconditioning Is Required to Prevent Opening of the Mitochondrial Permeability Transition Pore During Reperfusion, Circulation, vol.117, issue.21, pp.2761-2768, 2008. ,
DOI : 10.1161/CIRCULATIONAHA.107.755066
Statins are associated with a reduced incidence of perioperative mortality after coronary artery bypass graft surgery, Circulation, vol.110, issue.11, pp.45-49, 2004. ,
Atorvastatin and Myocardial Reperfusion Injury, Journal of Cardiovascular Pharmacology, vol.45, issue.3, pp.247-252, 2005. ,
DOI : 10.1097/01.fjc.0000154376.82445.06
Simvastatin Attenuates Oxidant-Induced Mitochondrial Dysfunction in Cardiac Myocytes, Circulation Research, vol.93, issue.8, pp.697-699, 2003. ,
DOI : 10.1161/01.RES.0000097262.21507.DF
Mitochondrial metabolism of reactive oxygen species, Biochemistry (Moscow), vol.1058, issue.13, pp.200-214, 2005. ,
DOI : 10.1007/s10541-005-0102-7
Measurement of superoxide-derived free radicals in the reperfused heart. Evidence for a free radical mechanism of reperfusion injury, J Biol Chem, vol.263, pp.1353-1357, 1988. ,
Age-dependent increase in hydrogen peroxide production by cardiac monoamine oxidase A in rats, American Journal of Physiology - Heart and Circulatory Physiology, vol.284, issue.4, pp.1460-1467, 2003. ,
DOI : 10.1152/ajpheart.00700.2002
Reactive oxygen species, but not Ca2+ overloading, trigger pH- and mitochondrial permeability transition-dependent death of adult rat myocytes after ischemia-reperfusion, AJP: Heart and Circulatory Physiology, vol.290, issue.5, pp.2024-2034, 2006. ,
DOI : 10.1152/ajpheart.00683.2005
Inhibition of Mitochondrial Permeability Transition Pore Opening by Ischemic Preconditioning Is Probably Mediated by Reduction of Oxidative Stress Rather Than Mitochondrial Protein Phosphorylation, Circulation Research, vol.102, issue.9, pp.1082-1090, 2008. ,
DOI : 10.1161/CIRCRESAHA.107.167072
Antioxidative properties of pyruvate and protection of the ischemic rat heart during cardioplegia mitochondrial membrane permeability, a pharmacological target for cardioprotection, J Cardiovasc Pharmacol . Curr Med Chem . Author manuscript Page, vol.34, issue.14, pp.651-659, 1999. ,
Are the Antioxidant Properties of Carvedilol Important for the Protection of Cardiac Mitochondria?, Current Vascular Pharmacology, vol.3, issue.2, pp.147-158, 2005. ,
DOI : 10.2174/1570161053586903
Protection of hearts from reperfusion injury by propofol is associated with inhibition of the mitochondrial permeability transition, Cardiovascular Research, vol.45, issue.2, pp.360-369, 2000. ,
DOI : 10.1016/S0008-6363(99)00365-X
Prevention of cell damage in ischaemia: novel molecular targets in mitochondria, Expert Opinion on Therapeutic Targets, vol.41, issue.2, pp.315-334, 2002. ,
DOI : 10.1038/415096a
A novel SOD-mimetic permeability transition inhibitor agent protects ischemic heart by inhibiting both apoptotic and necrotic cell death, Free Radical Biology and Medicine, vol.41, issue.5, pp.835-848, 2006. ,
DOI : 10.1016/j.freeradbiomed.2006.06.004
Antioxidant MCI-186 inhibits mitochondrial permeability transition pore and upregulates Bcl-2 expression, American Journal of Physiology - Heart and Circulatory Physiology, vol.285, issue.5, pp.2171-2178, 2003. ,
DOI : 10.1152/ajpheart.00143.2003
Oxidative Stress and Cardiovascular Injury: Part II: Animal and Human Studies, Circulation, vol.108, issue.17, pp.2034-2040, 2003. ,
DOI : 10.1161/01.CIR.0000093661.90582.c4
Targeting lipophilic cations to mitochondria, Biochimica et Biophysica Acta (BBA) - Bioenergetics, vol.1777, issue.7-8, pp.1028-1031 ,
DOI : 10.1016/j.bbabio.2008.03.029
Targeting an antioxidant to mitochondria decreases cardiac ischemia-reperfusion injury, The FASEB Journal, vol.19, issue.9, pp.1088-1095, 2005. ,
DOI : 10.1096/fj.05-3718com
Cell-permeable Peptide Antioxidants Targeted to Inner Mitochondrial Membrane inhibit Mitochondrial Swelling, Oxidative Cell Death, and Reperfusion Injury, Journal of Biological Chemistry, vol.279, issue.33, pp.34682-34690, 2004. ,
DOI : 10.1074/jbc.M402999200
Mitochondria-Targeted Cytoprotective Peptides for Ischemia???Reperfusion Injury, Antioxidants & Redox Signaling, vol.10, issue.3, pp.601-619, 2008. ,
DOI : 10.1089/ars.2007.1892
Oxidative Stress by Monoamine Oxidase Mediates Receptor-Independent Cardiomyocyte Apoptosis by Serotonin and Postischemic Myocardial Injury, Circulation, vol.112, issue.21, pp.3297-3305, 2005. ,
DOI : 10.1161/CIRCULATIONAHA.104.528133
Uncoupling: new approaches to an old problem of bioenergetics, Biochimica et Biophysica Acta (BBA) - Bioenergetics, vol.1363, issue.2, pp.100-124, 1998. ,
DOI : 10.1016/S0005-2728(97)00091-1
Effects of CCCP-induced mitochondrial uncoupling and cyclosporin A on cell volume, cell injury and preconditioning protection of isolated rabbit cardiomyocytes, Journal of Molecular and Cellular Cardiology, vol.35, issue.7, pp.749-759, 2003. ,
DOI : 10.1016/S0022-2828(03)00114-7
Mitochondrial uncoupling, with low concentration FCCP, induces ROS-dependent cardioprotection independent of KATP channel activation, Cardiovascular Research, vol.72, issue.2, pp.313-321, 2006. ,
DOI : 10.1016/j.cardiores.2006.07.019
Stable transfection of UCP1 confers resistance to hypoxia/reoxygenation in a heart-derived cell line, Journal of Molecular and Cellular Cardiology, vol.35, issue.7, pp.861-865, 2003. ,
DOI : 10.1016/S0022-2828(03)00147-0
Uncoupling Protein-2 Overexpression Inhibits Mitochondrial Death Pathway in Cardiomyocytes, Circulation Research, vol.93, issue.3, pp.192-200, 2003. ,
DOI : 10.1161/01.RES.0000085581.60197.4D
Uncoupling Proteins 2 and 3 Function in Concert to Augment Tolerance to Cardiac Ischemia, Journal of Biological Chemistry, vol.280, issue.39, pp.33470-33476, 2005. ,
DOI : 10.1074/jbc.M505258200