STUDIES ON THE PROPAGATION IN VITRO OF POLIOMYELITIS VIRUSES: IV. VIRAL MULTIPLICATION IN A STABLE STRAIN OF HUMAN MALIGNANT EPITHELIAL CELLS (STRAIN HELA) DERIVED FROM AN EPIDERMOID CARCINOMA OF THE CERVIX, Journal of Experimental Medicine, vol.97, issue.5, pp.695-710, 1953. ,
DOI : 10.1084/jem.97.5.695
Tumor stem cells derived from glioblastomas cultured in bFGF and EGF more closely mirror the phenotype and genotype of primary tumors than do serum-cultured cell lines, Cancer Cell, vol.9, issue.5, pp.391-403, 2006. ,
DOI : 10.1016/j.ccr.2006.03.030
Tissue oxygen tension and brain sensitivity to hypoxia, Respiration Physiology, vol.128, issue.3, pp.263-276, 2001. ,
DOI : 10.1016/S0034-5687(01)00306-1
Tumor Hypoxia: Definitions and Current Clinical, Biologic, and Molecular Aspects, JNCI Journal of the National Cancer Institute, vol.93, issue.4, pp.266-276, 2001. ,
DOI : 10.1093/jnci/93.4.266
URL : http://jnci.oxfordjournals.org/cgi/content/short/93/4/266
Influence of oxygen tension on CD133 phenotype in human glioma cell cultures, Cancer Letters, vol.258, issue.2, pp.286-290, 2007. ,
DOI : 10.1016/j.canlet.2007.09.012
URL : https://hal.archives-ouvertes.fr/inserm-00382779
Oxygen tension and cancer-cell culture: half a century of artifacts?, Med Sci, vol.24, pp.1093-1095, 2008. ,
URL : https://hal.archives-ouvertes.fr/inserm-00410499
PO2 Matters in Stem Cell Culture, Cell Stem Cell, vol.5, issue.3, pp.242-243, 2009. ,
DOI : 10.1016/j.stem.2009.08.009
URL : https://hal.archives-ouvertes.fr/inserm-00527773
???Pseudopalisading??? Necrosis in Glioblastoma: A Familiar Morphologic Feature That Links Vascular Pathology, Hypoxia, and Angiogenesis, Journal of Neuropathology & Experimental Neurology, vol.65, issue.6, pp.529-539, 2006. ,
DOI : 10.1097/00005072-200606000-00001
Vaso-occlusive and prothrombotic mechanisms associated with tumor hypoxia, necrosis, and accelerated growth in glioblastoma, Laboratory Investigation, vol.84, issue.4, pp.397-405, 2004. ,
DOI : 10.1038/labinvest.3700070
Significance of hemorrhage into brain tumors: clinicopathological study, Journal of Neurosurgery, vol.67, issue.6, pp.852-857, 1987. ,
DOI : 10.3171/jns.1987.67.6.0852
Transdifferentiation of glioblastoma cells into vascular endothelial cells, Proceedings of the National Academy of Sciences, vol.108, issue.11, pp.4274-4280, 2011. ,
DOI : 10.1073/pnas.1016030108
Tumour vascularization via endothelial differentiation of glioblastoma stem-like cells, Nature, vol.200, issue.7325, pp.824-828, 2010. ,
DOI : 10.1038/nature09557
Endothelial-like cells derived directly from human tumor xenografts, International Journal of Cancer, vol.34, issue.Suppl 1, pp.2268-2278, 2010. ,
DOI : 10.1002/ijc.25251
Glioblastoma cells incorporate into tumor vasculature and contribute to vascular radioresistance, International Journal of Cancer, vol.87, issue.9, pp.2063-2075, 2010. ,
DOI : 10.1002/ijc.25249
URL : http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2932815
Glioblastoma stem-like cells give rise to tumour endothelium, Nature, vol.459, issue.7325, pp.829-833, 2010. ,
DOI : 10.1038/nature09624
VE-cadherin: at the front, center, and sides of endothelial cell organization and function, Current Opinion in Cell Biology, vol.22, issue.5, pp.651-658, 2010. ,
DOI : 10.1016/j.ceb.2010.07.006
Filamin B Plays a Key Role in Vascular Endothelial Growth Factor-induced Endothelial Cell Motility through Its Interaction with Rac-1 and Vav-2, Journal of Biological Chemistry, vol.285, issue.14, pp.10748-10760, 2010. ,
DOI : 10.1074/jbc.M109.062984
Pseudopalisades in Glioblastoma Are Hypoxic, Express Extracellular Matrix Proteases, and Are Formed by an Actively Migrating Cell Population, Cancer Research, vol.64, issue.3, pp.920-927, 2004. ,
DOI : 10.1158/0008-5472.CAN-03-2073
Neuropathology for the neuroradiologist: palisades and pseudopalisades, AJNR Am J Neuroradiol, vol.27, pp.2037-2041, 2006. ,
Hypoxia-Inducible Factors in Physiology and Medicine, Cell, vol.148, issue.3, pp.399-408, 2012. ,
DOI : 10.1016/j.cell.2012.01.021
Hypoxia-inducible factor 1 levels vary exponentially over a physiologically relevant range of O2 tension, Am J Physiol, vol.271, pp.1172-1180, 1996. ,
The Oxygen Sensor Factor-Inhibiting Hypoxia-Inducible Factor-1 Controls Expression of Distinct Genes through the Bifunctional Transcriptional Character of Hypoxia-Inducible Factor-1??, Cancer Research, vol.66, issue.7, pp.3688-3698, 2006. ,
DOI : 10.1158/0008-5472.CAN-05-4564
URL : https://hal.archives-ouvertes.fr/hal-00321066
Gene regulation in response to graded hypoxia: The non-redundant roles of the oxygen sensors and FIH in the HIF pathway, Journal of Theoretical Biology, vol.259, issue.2, pp.304-316, 2009. ,
DOI : 10.1016/j.jtbi.2009.03.009
URL : https://hal.archives-ouvertes.fr/hal-00554587
In vitro expansion of human glioblastoma cells at non-physiological oxygen tension irreversibly alters subsequent in vivo aggressiveness and AC133 expression, Int J Oncol, vol.40, pp.1220-1229, 2012. ,
URL : https://hal.archives-ouvertes.fr/inserm-00733941
Vascular endothelial tyrosine phosphatase (VE-PTP)-null mice undergo vasculogenesis but die embryonically because of defects in angiogenesis, Proceedings of the National Academy of Sciences, vol.104, issue.9, pp.3243-3248, 2007. ,
DOI : 10.1073/pnas.0611510104
Vascular endothelial cell-specific phosphotyrosine phosphatase (VE-PTP) activity is required for blood vessel development, Blood, vol.107, issue.12, pp.4754-4762, 2006. ,
DOI : 10.1182/blood-2006-01-0141
URL : http://nbn-resolving.de/urn/resolver.pl?urn=urn:nbn:de:bvb:12-bsb00060029-7
VE-PTP and VE-cadherin ectodomains interact to facilitate regulation of phosphorylation and cell contacts, The EMBO Journal, vol.21, issue.18, pp.4885-4895, 2002. ,
DOI : 10.1093/emboj/cdf497
VE-PTP controls blood vessel development by balancing Tie-2 activity, The Journal of Cell Biology, vol.244, issue.4, pp.657-671, 2009. ,
DOI : 10.1016/j.immuni.2007.11.024
URL : http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2711575
Promotion of cell spreading and migration by vascular endothelial-protein tyrosine phosphatase (VE-PTP) in cooperation with integrins, Journal of Cellular Physiology, vol.13, pp.195-204, 2010. ,
DOI : 10.1002/jcp.22122
Differentiation and expansion of endothelial cells from human bone marrow CD133+ cells, British Journal of Haematology, vol.90, issue.1, pp.186-194, 2001. ,
DOI : 10.1126/science.285.5433.1553
Insulin-like growth factor-binding protein-3 promotes transforming growth factor-{beta}1-mediated epithelial-tomesenchymal transition and motility in transformed human esophageal cells. Carcinogenesis, pp.1344-1353, 2010. ,
Increased concentrations of transforming growth factor ??1 and ??2 in the plasma of patients with glioblastoma, Journal of Neuro-Oncology, vol.72, issue.1, pp.61-65, 2006. ,
DOI : 10.1007/s11060-005-9116-7
Existence of tumor-derived endothelial cells suggests an additional role for endothelial-to-mesenchymal transition in tumor progression, International Journal of Cancer, vol.128, issue.6, pp.1502-1503, 2011. ,
DOI : 10.1002/ijc.25446
URL : https://hal.archives-ouvertes.fr/inserm-00498732